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Molecular mechanism of C-peptide in the pathogenesis of T2DM-induced atherosclerosis |
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Exercise as an anti-inflammatory therapy for rheumatic diseases-myokine regulation |
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2016.03.25 |
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Æ÷½ºÅÍ_-_2016.03.25_(±Ý)_·¦¹ÌÆÃ.ppt |
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Cereblon in multiple myeloma |
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Computational fluid dynamics modelling in cardiovascular medicine |
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2016.03.18 |
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Æ÷½ºÅÍ_-_2016.03.18_(±Ý)_·¦¹ÌÆÃ.ppt |
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Proteomic profiling and mechanistic differences in contractility of the left and right ventricles in rat heart |
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Role of Interferon Regulatory Factor 4 in the Regulation of Pathological Cardiac Hypertrophy |
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2016.03.04 |
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Æ÷½ºÅÍ_-_2016.03.04_(±Ý)_·¦¹ÌÆÃ.ppt |
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Echinochrome A suppress atherosclerosis progression |
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The role of endothelial mechanosensitive genes in atherosclerosis and omics approaches |
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2016.02.26 |
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Æ÷½ºÅÍ_-_2016.02.26_(±Ý)_·¦¹ÌÆÃ.ppt |
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Regulatory mechanism of CO2 in mouse heart and mitochondria function |
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Percutaneous carbon dioxide mist treatment has protective effects in experimental myocardial infarction |
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2016.02.19 |
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Æ÷½ºÅÍ_-_2016.02.19_(±Ý)_·¦¹ÌÆÃ.ppt |
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Anatomical modeling of the coronary artery |
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Exercise-stimulated interleukin-15 is controlled by AMPK and regulates skin metabolism and aging |
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2016.02.12 |
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Æ÷½ºÅÍ_-_2016.02.12_(±Ý)_·¦¹ÌÆÃ.ppt |
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The evaluation of the associations between coronary artery atherosclerosis and left ventricle using computational fluid dynamics |
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Lack of glutathione peroxidase-1 facilitates a pro-inflammatory and activated vascular endothelium |
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2016.02.05 |
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Æ÷½ºÅÍ_-_2016.02.05_(±Ý)_·¦¹ÌÆÃ.ppt |
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Mitochondrial DNA, mitochondrial dysfunction, and cardiac manifestations |
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2016.01.29 |
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Æ÷½ºÅÍ_-_2016.01.29_(±Ý)_·¦¹ÌÆÃ.ppt |
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A mathematical model of the beta-adrenergic signaling alterations in mouse ventricular myocytes in myocardial infarction. |
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Mitochondria as a Drug Target in Ischemic Heart Disease and Cardiomyopathy. |
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2016.01.22 |
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Æ÷½ºÅÍ_-_2016.01.22_(±Ý)_·¦¹ÌÆÃ.ppt |
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Analyzing whole-cell protocols with computer simulation. |
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Knockout of p21-activated kinase-1 attenuates exercise-induced cardiac remodelling through altered calcineurin signaling. |
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2016.01.15 |
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Æ÷½ºÅÍ_-_2016.01.15_(±Ý)_·¦¹ÌÆÃ.ppt |
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